Special Precautions/Comments:

Additional Information:

Consultant Approved Referral Test

Heparin Induced Thrombocytopenia (HIT) is caused by the development of IgG antibodies directed against a complex of platelet factor 4 (PF4) and heparin. The optimal concentration of heparin to produce conditions that favour the development of HIT are thought to be associated with prophylactic rather than therapeutic doses of heparin. The IgG/PF4/heparin complexes bind to and activate platelets through their Fc receptors and may also generate thrombin by other actions resulting in a prothrombotic condition that is associated with venous and arterial thrombosis.
Consultant Approved Referral Test

Heparin Induced Thrombocytopenia (HIT) is caused by the development of IgG antibodies directed against a complex of platelet factor 4 (PF4) and heparin. The optimal concentration of heparin to produce conditions that favour the development of HIT are thought to be associated with prophylactic rather than therapeutic doses of heparin. The IgG/PF4/heparin complexes bind to and activate platelets through their Fc receptors and may also generate thrombin by other actions resulting in a prothrombotic condition that is associated with venous and arterial thrombosis.
Consultant Approved Referral Test

Heparin Induced Thrombocytopenia (HIT) is caused by the development of IgG antibodies directed against a complex of platelet factor 4 (PF4) and heparin. The optimal concentration of heparin to produce conditions that favour the development of HIT are thought to be associated with prophylactic rather than therapeutic doses of heparin. The IgG/PF4/heparin complexes bind to and activate platelets through their Fc receptors and may also generate thrombin by other actions resulting in a prothrombotic condition that is associated with venous and arterial thrombosis.
Consultant Approved Referral Test

Heparin Induced Thrombocytopenia (HIT) is caused by the development of IgG antibodies directed against a complex of platelet factor 4 (PF4) and heparin. The optimal concentration of heparin to produce conditions that favour the development of HIT are thought to be associated with prophylactic rather than therapeutic doses of heparin. The IgG/PF4/heparin complexes bind to and activate platelets through their Fc receptors and may also generate thrombin by other actions resulting in a prothrombotic condition that is associated with venous and arterial thrombosis.